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. 2018 Sep 27;9:574. doi: 10.3389/fendo.2018.00574

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Copyright © 2018 Liu, He, Ning, Luo, Li, Zhang, Chen and Du.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Physiological functions of LepA in Nile tilapia. (A) Accumulated food intake rate, (n = 3, 5 fish each parallel). (B) Plasma TG (n = 6), NEFA (n = 6), glucose (n = 7), glucagon and insulin (INS) levels (n = 5). (C) Liver TG content (n = 6). (D) Total β-oxidation of [1-¹⁴C] palmitate in the liver homogenates (n = 6). (E) Liver glycogen content (n = 6). (F) Reservation of [1-¹⁴C] D-glucose in body (n = 6). (G) LepR, SOCS3 and NPY mRNA in hypothalamus (n = 6). (H) ATGL and apoE mRNA in liver (n = 6). (I) Glut2 mRNA in liver (n = 6). *p < 0.05 and ^**p < 0.01. Data shown as means ± SEM.