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. 2018 Sep 27;42(6):3027–3036. doi: 10.3892/ijmm.2018.3902

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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miR-214 expression is inversely correlated with PSMD10 expression in PTC tissues. (A) The phosphorylation activities of AKT and GSK-3β, and the protein expression levels of β-catenin, were examined in PTC cells by western blotting. PSMD10 knockdown repressed GSK-3β/β-catenin and AKT signaling in PTC cells. (B) Correlation analysis of the miR-214 expression levels and the PSMD10 expression levels in 30 pairs of human PTC tissues. PSMD10, proteasome 26S subunit non-ATPase 10; PTC, papillary thyroid carcinoma; GSK, glycogen synthase kinase; si, small interfering; Ctrl, control; p-, phosphorylated.