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. 2018 Oct 19;9:779. doi: 10.3389/fneur.2018.00779

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Copyright © 2018 Wen, Wang, Li, Jiang, Sun and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effect of endogenous H₂S on cerebrovascular dysfunction injured by cerebral I/R (mean ± SD, n = 8). (A) Original tracings of U46619-, ACh-, vinpocetine-, and NaHS-induced responses of MCA derived from CSE knocked down rats after MCAO. (B) The effect of CSE knockdown and NaHS on U46619-meditated contraction. (C) The effect of CSE knockdown on and NaHS- or ACh-meditated relaxation. (D) The effect of CSE knockdown and NaHS on vinpocetine-meditated relaxation. ^**P < 0.01 vs. sham, ^##P < 0.01 vs. MCAO.