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. 2018 Oct 25;18:1037. doi: 10.1186/s12885-018-4935-z

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Dynamic changes of LVEF, GLS, GCS, RVLS and GLSRs at different time points (T0-T2). Multi-line graph showing the relationship between LVEF% (purple line), GLS% (blue line), RVLS%(green line), GCS% (red line) and GCSRs (orange line), at three different time points: T0, T1, T2. Notice that there is significant change between T0 and T1 for right ventricular longitudinal strain (RVLS), global circumferential strain (GCS), global longitudinal strain (GLS), and global circumferential systolic strain rate (GCSRs) at T1 that can predict the development of cardiac toxicity at T2