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. 2018 Oct 26;9(11):1095. doi: 10.1038/s41419-018-1079-7

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — Representative sections of WT and Mkk4/Mkk7-deficient retinas stained with specific cell markers show clear disorganization of amacrine cells (a), slight defects in horizontal cells (b), and abnormal distribution and morphology of bipolar cells (c) and Müller glia (d) in retinas of Mkk4/Mkk7 double-deficient mutants. Merged images with DAPI (blue) are shown below. N ≥ 4 per genotype. e Mkk4/Mkk7 double-deficient flat mounts further display clumping and dendritic fasciculation of dopaminergic amacrine cells. N ≥ 4 per genotype. Scale bars: 100 μm