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. 2018 Oct 26;18:259. doi: 10.1186/s12870-018-1485-0

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Constitutive TaBI-1.1 expression in atbi1–2 rescues the deficiency of atbi1–2 in heat tolerance. a The phenotypes of atbi1–2, Col-0, 35S::TaBI-1.1/atbi1–2#1, and 35S::TaBI-1.1/atbi1–2#2 under heat stress. b The survival rates of atbi1–2, Col-0, 35S::TaBI-1.1/atbi1–2#1, and 35S::TaBI-1.1/atbi1–2#2 under heat stress. The results are shown as the means±SD of three biological replicates. Error bars indicate the SD. c The relative conductivity of atbi1–2, Col-0, 35S::TaBI-1.1/atbi1–2#1, and 35S::TaBI-1.1/atbi1–2#2 after heat treatment. The results are shown as the means±SD of six biological replicates. Error bars indicate the SD. d Hypocotyl elongation in atbi1–2, Col-0, 35S::TaBI-1.1/atbi1–2#1, and 35S::TaBI-1.1/atbi1–2#2 after heat treatment. e Statistical analysis of hypocotyl elongation in atbi1–2, Col-0, 35S::TaBI-1.1/atbi1–2#1, and 35S::TaBI-1.1/atbi1–2#2 after heat treatment. The results are shown as the means±SD of 36 biological replicates. Error bars indicate the SD. All the asterisks in the figure (* and **) indicate significant differences (P < 0.05 and P < 0.01, respectively) compared with Col-0 (Student’s t-test)