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. 2018 Oct 9;16(10):374. doi: 10.3390/md16100374

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© 2018 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Inhibitory effect of MES on the reactive oxygen species generation, lipid peroxidation, and GSH depletion in t-BHP-treated HepG2 cells. HepG2 cells were treated with MES (0.25~1.0 μg/mL) and t-BHP. (A) ROS were measured by a fluorescent probe, DCFH-DA. (B) Lipid peroxidation was measured by using a TBARS assay. (C) The GSH level was measured by fluorescent probe, CMF-DA. The data are means ± SD of three independent experiments. # p < 0.05 indicates significant differences from the control group. * p < 0.05 indicates significant differences from the t-BHP treatment group.

Inhibitory effect of MES on the reactive oxygen species generation, lipid peroxidation, and GSH depletion in t-BHP-treated HepG2 cells. HepG2 cells were treated with MES (0.25~1.0 μg/mL) and t-BHP. (A) ROS were measured by a fluorescent probe, DCFH-DA. (B) Lipid peroxidation was measured by using a TBARS assay. (C) The GSH level was measured by fluorescent probe, CMF-DA. The data are means ± SD of three independent experiments. # p < 0.05 indicates significant differences from the control group. * p < 0.05 indicates significant differences from the t-BHP treatment group.