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. 2018 Oct 21;19(10):3267. doi: 10.3390/ijms19103267

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© 2018 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Potential molecular therapies targeting mTOR-dependent metabolic reprogramming in cancer cells. xCT: amino acid transport system Xc-; NF-κB: nuclear factor-kappa B; RTK: receptor tyrosine kinase; JAK: Janus kinase; STAT: signal transducers and activators of transcription; ERK: extracellular signal-regulated kinase; S6K1: ribosomal protein S6 kinase 1; 4EBP1: eukaryotic translation initiation factor 4E-binding protein 1; LKB1: liver kinase B1; AMPK: adenosine monophosphate-activated protein kinase; SREBP-1: sterol regulatory element-binding protein 1; LDLR: low density lipoprotein receptor; IDOL: inducible degrader of LDLR; ABCA1: ATP-binding cassette protein A1; LXR: liver X receptor.