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. 2018 Sep 28;10(10):398. doi: 10.3390/toxins10100398

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© 2018 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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The role of the PI3K/Akt/m TOR signaling pathway in autophagy induced by ZEA. (A) The cells in the logarithmic phase were treated with different concentrations of ZEA for 24 h, Western-Blot detected the expression of key proteins phosphorylation levels in the PI3K/Akt/m TOR signaling pathway (* p < 0.05, ** p < 0.01); (B,C) Expression levels of p-AKT/AKT, p m TOR/m TOR, and LC3II/LC3I after adding PI3K inhibitors (LY294002) in the experiments (** p < 0.01); (D) Effects of PI3K inhibitor on point phenomenon of SCs treated with the ZEA.

The role of the PI3K/Akt/m TOR signaling pathway in autophagy induced by ZEA. (A) The cells in the logarithmic phase were treated with different concentrations of ZEA for 24 h, Western-Blot detected the expression of key proteins phosphorylation levels in the PI3K/Akt/m TOR signaling pathway (* p < 0.05, ** p < 0.01); (B,C) Expression levels of p-AKT/AKT, p m TOR/m TOR, and LC3II/LC3I after adding PI3K inhibitors (LY294002) in the experiments (** p < 0.01); (D) Effects of PI3K inhibitor on point phenomenon of SCs treated with the ZEA.