Table 2. Sterility and embryonic lethality in oma-2 and oma-1; oma-2 mutant strains at 20°.
Genotype | Embryos laida | Dead embryos, % |
---|---|---|
oma-2(cp145) | 314 ± 48 (n = 6) | 0.6 (n = 1793) |
oma-2(tn1760) | 306 ± 39 (n = 6) | 0.6 (n = 1835) |
oma-2(tn1764) | 300 ± 45 (n = 6) | 2.1 (n = 1797) |
oma-2(tn1764) lon-3(e2175) sel-10(ar41) | 288 ± 26 (n = 6) | 1.1 (n = 1727) |
oma-1(zu405te33) | 261 ± 18 (n = 6) | 0.8 (n = 1568) |
oma-1(zu405te33); oma-2(te51) M+Z–b | 0 (n > 21)c | NA |
oma-1(zu405te33); oma-2(cp145) | 212 ± 29 (n = 12) | 12.3d (n = 2516) |
oma-1(zu405te33); oma-2(tn1760) | 224 ± 35 (n = 6) | 60.3 (n = 1341) |
oma-1(zu405te33); oma-2(tn1764) M+Z–b,e | 249 ± 29 (n = 5) | 100 (n > 1256) |
oma-1(zu405te33); oma-2(tn1764) lon-3(e2175) sel-10(ar41) M+Z–b,f | 246 ± 32 (n = 6) | 89.4 (n = 1476) |
oma-1(zu405te33); oma-2(tn1764) lon-3(e2175) sel-10(ar41) | 196 ± 56 (n = 6) | 84.9 (n = 1175) |
Average number of embryos laid per worm ± SD.
M+Z– animals were the progeny of nT1[qIs51] balancer-containing parents, which are heterozygous for both oma-1 and oma-2. All other animals were the progeny of parents of the listed genotype.
Sterile, with a defect in meiotic maturation as described by Detwiler et al. (2001).
Percent embryo lethality was variable among the 12 parents analyzed; it ranged between 6 and 35%.
These animals lay many eggs, none of which hatch (n = 30).
These animals lay many eggs, some of which hatch (n = 24).