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. 2018 Nov 8;13(11):e0207162. doi: 10.1371/journal.pone.0207162

Table 1. Baseline characteristics of included patients.

No Hospitalization due to decompensation at follow-up
N = 287
Hospitalization at follow-up due to decompensation
N = 51
Age (years); [SD] 59.40 [12.09] 61.45 [10.99]
Coffee (cups/day); [SD] 1.65 [1.65] 0.47 [0.92]
Diabetes (presence); N [%] 63 [22] 21 [41]
BMI (kg/m2); [SD] 27.10 [4.95] 26.20 [5.81]
MELD Score; [SD] 9.68 [3.83] 13.76 [4.39]
Creatinin (mg/dl); [SD] 0.86 [0.29] 1.14 [0.83]
Albumin (g/dl); [SD] 4.12 [0.62] 3.25 [0.59]
Bilirubin (mg/dl); [SD] 1.33 [1.45] 2.27 [1.68]
ALT (U/L); [SD] 37.59 [43.70] 34.62 [24.60]
yGT (U/L); [SD] 111.3 [144.82] 205.66 [296.35]
Leukocyte (/nl); [SD] 6.03 [2.23] 5.66 [2.74]
Hb (mg/dl); [SD] 13.42 [1.91] 11.15 [2.31]
Platelets (/nl); [SD] 141.05 [71.21] 122.96 [78.65]
25(OH)D3 (ng/ml); [SD] 27.95 [17.81] 21.24 [18.75]
IL-6 (pg/mL); [SD] 3.61 [4.93] 1.52 [34.03]
I-FABP (pg/mL);[SD] 1155.5 [928.16] 1973.2 [2050.9]
sCD14 (ng/mL); [SD] 2.15 [0.66] 2.58 [0.59]
Ascites at baseline, N [%] 26 [9] 27 [52]
Hepatic encephalopathy at baseline, N [%] 1 [0.35] 2 [4]
History of ascites, N [%] 105 [37] 36 [71]
History of hepatic decompensation, N [%] 13 [45] 44 [86]
Liver cirrhosis etiology of entire cohort n,(%)
HCV 117 (35)
HBV 53 (16)
Alcoholic 103 (30)
NASH 33 (10)
Other 32 (11)

ALT, alanine aminotransferase; BMI, body mass index; range; γGT, γ-glutamyl transferase; Hb, hemoglobin; I-FABP, intestinal fatty acid binding protein; MELD, model of end-stage liver disease.