Skip to main content
. Author manuscript; available in PMC: 2019 Nov 1.
Published in final edited form as: Thorax. 2018 Jun 13;73(11):1041–1048. doi: 10.1136/thoraxjnl-2017-211383

Table 3:

Associations between SHS exposure (defined using self-report and measured plasma cotinine (>0.05 ng/mL or above the limit of quantification if higher than 0.05 ng/mL)) and asthma outcomes.

Asthma outcomes OR (95% CI)
Self-reported exposure* Plasma cotinine determined exposure
Asthma exacerbations in the previous 12 months 1.08 (0.75 – 1.57) 1.40 (1.03 – 1.89)
 Hospitalizations 1.77 (0.79 – 3.95) 1.26 (0.60 – 2.67)
 ER visits 1.21 (0.70 – 2.09) 1.07 (0.65 – 1.75)
 Oral steroid prescription 1.00 (0.71 – 1.41) 1.54 (1.18 – 2.04)
Asthma control (binary outcome) 1.62 (1.09 – 2.38) 1.53 (1.12 – 2.13)
 Not well controlled vs controlled 1.43 (0.93 – 2.21) 1.39 (0.98 – 1.98)
 Very poorly controlled vs controlled 1.79 (1.18 – 2.70) 1.67 (1.18 – 2.36)
*

Self-reported exposure to SHS was determined as any vs no reported current smokers in the household.

Asthma exacerbations assessed as a combined variable for any exacerbations (hospitalizations, ER visits, prescription of oral steroids) vs none

Asthma control assessed as binary outcome with a combined “not well controlled” and “very poorly controlled” category compared to controlled asthma