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. 2018 Oct 21;20:402–413. doi: 10.1016/j.redox.2018.10.005

Fig. 2.

Fig. 2

Treatment of animals with an antisense oligonucleotide against SOD2 prevents the exercise-induced increase in SOD2 activity and protein abundance in the diaphragm while attenuating exercise protection against VIDD and abolishing exercise protection against atrophy. A) ELISA of SOD2 protein abundance in diaphragm, n = 9/group. B) SOD2 activity in diaphragm, n = 8/group. Values are mean percent change of control ± SE. # p < 0.05 different from MVEx; δ p < 0.05 different from all groups. C) Diaphragm specific force, n = 10/group. Values are mean ± SE. * p < 0.05 MV different from CON, # p < 0.05 MV different from MVEx; θ p < 0.05 MVEx-AS different from CON; δ p < 0.05 MV different from all groups. D) Diaphragm cross-sectional area. Values are mean ± SE. * p < 0.05 different from CON, # p < 0.05 different from MVEx.