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. 2018 Nov 12;19:194. doi: 10.1186/s13059-018-1567-1

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Association between expression of the Complement C4A gene in fetal brain and genetic risk for schizophrenia. a Effect sizes of SNPs from schizophrenia GWAS [36] plotted against their effect sizes as fetal brain cis-eQTL for C4A. The triangular data point indicates the variant with the lowest P-SMR. Error bars are the standard errors of SNP effects. b Homozygotes for the schizophrenia-associated C-allele of rs9267544 exhibit increased expression of C4A in fetal brain compared with heterozygotes for this SNP. Error bars represent standard errors