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. 2018 Nov 12;37:274. doi: 10.1186/s13046-018-0947-4

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© The Author(s). 2018

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — LAT2 is upregulated in pancreatic cancer. a The expression level of LAT2 in 71 pancreatic cancer tissue samples was higher than that in the matched paracancerous tissues (*, P < 0.05). b The Wilcoxon signed rank test was applied to compare the difference in LAT2 expression between the cancer and paracancerous tissues. c The expression of LAT2 in pancreatic cancer tissues was evaluated by immunohistochemistry. d The expression of LAT2 in pancreatic paracancerous tissues was evaluated by immunohistochemistry. e Kaplan-Meier survival analysis indicated that the overall survival time of the high-LAT2-level group is shorter than that of the low-LAT2-level group of patients with pancreatic cancer (P = 0.0154)