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. 2018 Nov 8;8:503. doi: 10.3389/fonc.2018.00503

Table 1.

Radiation-induced alterations to the GBM micro-environment.

Biological process Consequence RT-effect References
EXTRACELLULAR MATRIX COMPOSITION AND BIOSYNTHESIS
Collagen Migration and Invasion +/Up (38, 45, 46)
Tenascin C Tumor proliferation, Invasion +/Up (28, 29, 47)
Hyaluronin Invasion +/Up (48, 49)
Brevican Migration, Invasion +/Up (25, 30, 50)
Vitronectin Survival, Migration, Inflammation +/Up (26)
MDA-9/Syntenin Metastasis, tumor progression +/Up (51)
LOX Migration +/Up (52)
ECM-GLIOMA CELL (LIGAND-RECEPTOR) INTERACTION
DDR-1, ICAM-1, α5β1, αvβ3 Migration, Invasion +/Up (31, 5355)
ECM DEGRADATION
MMPs Invasion +/Up (5659)
TIMP Angiogenesis, Metastasis +/Up (60, 61)
TUMOR CELL ADAPTATION MECHANISMS
Oxygen tension: HIF-1 Hypoxia, malignancy +/Up (62)
Metabolism: ATP, NAD Proliferation +/Up (63)
Anti-apoptosis: BCL2/BAX Migration, invasiveness +/Up (55)
Redox regulation (ROS/RNS, NOX4) Senescence, inflammation +/Up (6466)
Angiogenesis (VEGF, Ang) Angiogenesis +/Up (67)
Inflammation (Cytokines, Chemokines, Chemokine receptors) Tumor proliferation, migration, invasion +/Up (6872)
Glia activation (MHC, CD68, GFAP) proliferation +/Up (70, 7375)
Neurogenesis (NSC) Cognitive decline -/Impaired (76)

MDA-9, Melanoma differentiation-associated gene-9; LOX, Lysl oxidase; DDR-1, Discoidin domain receptor-1; ICAM-1, Intercellular Adhesion Molecule 1; Integrins α5β1; Integrin, αvβ3; MMPs, Matrix metalloproteinases; TIMP, Tissue inhibitor of matrix metalloproteinase; NSC, Neural stem cell; HIF-1, Hypoxia-inducible factor 1; VEGF, Vascular endothelial growth factor; Ang, Angiotensins; CALR, Calreticulin; HGMB1, High mobility group box 1 protein; NOX4, NADPH oxidase 4; ROS, Reactive oxygen species; RNS, Reactive nitrogen species; MHC, Major histocompatibility complex; ATP, Adenosine triphosphate; GFAP, Glial acidic fibrillary protein; NAD, Nicotinamide adenine dinucleotide; CD, Cluster of differentiation; BCL2, B-cell lymphoma-2; BAX, BCL2 Associated X. Cytokines: Tumor necrosis factor-α, TNF- α; Transforming growth factor-β, TGF-β, IL, Interleukins (IL-6, IL-8, IL-1β). Chemokines: CX3C family (CX3CL1, Fractalkine), CCL family (CCL2, CCR3, CCL7, CCL8, CCL12), CXC family (CXCL4, CXCL12/stromal cell-derived factor 1, SDF1). Chemokine receptors: CC-chemokine receptor family (CCR1, CCR2). RT-effects: +, positive; Up-upregulation/increased; –, negative.