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. 2018 Oct 15;18(6):5669–5682. doi: 10.3892/mmr.2018.9557

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Verification of the expression of significantly dysregulated genes by reverse transcription-quantitative polymerase chain reaction. The relative expression levels of (A) five long noncoding RNAs, (B) six circular RNAs and (C) five mRNAs were detected in N and H groups. Data are presented as mean ± SD, *P<0.05. ASXL1, ASXL transcriptional regulator 1; COL1A1, collagen type I A1 chain; CTGF, connective tissue growth factor; H, hypertrophic scar tissues; HIPK3, homeodomain interacting protein kinase 3; N, normal tissues; TGF-β3, transforming growth factor β3.

Figure 2. — Verification of the expression of significantly dysregulated genes by reverse transcription-quantitative polymerase chain reaction. The relative expression levels of (A) five long noncoding RNAs, (B) six circular RNAs and (C) five mRNAs were detected in N and H groups. Data are presented as mean ± SD, *P<0.05. ASXL1, ASXL transcriptional regulator 1; COL1A1, collagen type I A1 chain; CTGF, connective tissue growth factor; H, hypertrophic scar tissues; HIPK3, homeodomain interacting protein kinase 3; N, normal tissues; TGF-β3, transforming growth factor β3.