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. 2018 Nov 14;13:205. doi: 10.1186/s13023-018-0938-8

Table 8.

Distribution of MEN1-related tumours/lesions with respect to MEN1 type of mutations

Tumour/lesion (n.) FS
n. (%)
MS
n. (%)
NS
n. (%)
SP
n. (%)
FS + MS
n. (%)
Intragenic large deletions 11q13 familial haplotype No identified MEN1 mutation*
PHPT (139) 47 (100%) 37 (100%) 17 (94.44%) 22 (91.67%) 3 (75%) 4 (100%) 2 (100%) 7 (70%)
 Symptomatic (54) 12 (25.53%) 18 (48.65%) 8 (44.44%) 9 (37.50%) 1 (25%) 3 (75%) 1 (50%) 2 (20%)
 Asymptomatic (85) 35 (74.47%) 19 (51.35%) 9 (50%) 13 (54.17%) 2 (50%) 1 (25%) 1 (50%) 5 (50%)
GEP-NETs (86) 32 (68.09%) 16 (43.24%) 12 (66.67%) 13 (54.17%) 2 (50%) 4 (100%) 2 (100%) 5 (50%)
 pNFTs (28) 12 (25.53%) 6 (16.22%) 3 (16.67%) 4 (16.67%) 1 (25%) 1 (25%) 0 1 (10%)
 PPoma (1) 0 1 (2.70%) 0 0 0 0 0 0
 pNFT-gastric NF NET (1) 0 0 0 1 (4.17%) 0 0 0 0
 Gastrinomas (28) 7 (14.89%) 3 (8.11%) 5 (27.78%) 7 (29.17%) 1 (25%) 2 (50%) 0 3 (30%)
 Insulinomas (13) 6 (12.77%) 2 (5.41%) 3 (16.67%) 1 (4.17%) 0 0 0 1 (10%)
 VIPomas (2) 1 (2.13%) 1 (2.70%) 0 0 0 0 0 0
 Gastrinoma-insulinoma (3) 2 (4.26%) 1 (2.70%) 0 0 0 0 0 0
 Gastrinoma-glucagonoma (1) 0 1 (2.70%) 0 0 0 0 0 0
 pNFTs-gastrinoma (9) 4 (8.51%) 1 (2.70%) 1 (5.56%) 0 0 1 (25%) 2 (100%) 0
Pituitary adenomas (75) 24 (51.06%) 19 (51.35%) 10 (55.56%) 11 (45.83%) 3 (75%) 1 (25%) 1 (50%) 6 (60%)
 NFAs (12) 4 (8.51%) 2 (5.41%) 3 (16.67%) 2 (8.33%) 0 0 0 1 (10%)
 PRLomas (59) 18 (38.30%) 17 (45.95%) 6 (33.33%) 9 (24.32%) 3 (75%) 1 (25%) 1 (50%) 4 (40%)
 Corticotropinomas (3) 1 (2.13%) 0 1 (5.56%) 0 0 0 0 1 (10%)
 PRLoma-somatotropinoma (1) 1 (2.13%) 0 0 0 0 0 0 0
Bronchopulmonary carcinoids (17) 5 (10.64%) 3 (8.11%) 2 (11.11%) 6 (25%) 0 0 0 1 (10%)
Skin lesions
 Lipomas (37) 16 (34.04%) 5 (13.51%) 2 (11.11%) 7 (29.17%) 3 (75%) 3 (75%) 0 1 (10%)
 Angiomas/angiofibromas/fibromas (13) 3 (6.38%) 5 (13.51%) 2 (11.11%) 1 (4.17%) 1 (25%) 1 (25%) 0 0
Adrenal gland tumours/lesions (27) 9 (19.15%) 8 (21.62%) 3 (16.67%) 3 (12.5%) 2 (50%) 1 (25%) 0 1 (10%)

Footnotes: N. number, FS Frameshift, MS Missense, NS nonsense, SP Splicing site, PHPT Primary hyperparathyroidism, GEP-NETs Gastro-entero-pancreatic neuroendocrine tumours, pNFTs Pancreatic non-functioning tumours, NF NET Non-functioning neuroendocrine tumours, VIP Vasoactive intestinal peptide, NFAs Non-functioning adenomas

Percentages were calculated with respect to the number of any mutation type in symptomatic patients (47 frameshifts, 18 nonsense, 37 missense, 24 splicing site, 4 double mutation, 4 large intragenic deletions and 2 11q13 predisposing familial haplotype)

*Patients analysed only by PCR-based Sanger’s sequencing