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. 2018 Nov 16;9:4820. doi: 10.1038/s41467-018-07226-6

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Responses to phytochemical toxins and insecticides (SCD-d6AE relative to SCD). The SCD-d6AE strain was derived from SCD by knocking out the CYP6AE cluster. Increased folds in susceptibility were calculated as LC₅₀ of SCD/LC₅₀ of SCD-d6AE. LC₅₀ values were considered significantly different if their fiducial limits did not overlap, and asterisks indicate statistical significance of LC₅₀s between the two strains. Phytochemical toxins: xanthotoxin, gossypol acetate, 2-tridecanone, nicotine, and coumarin. Insecticides: esfenvalerate, indoxacarb, emamectin benzoate, and chlorantraniliprole