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. 2018 Nov 19;9(11):208. doi: 10.1038/s41424-018-0076-6

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Patients with defined achalasia type I, type II, type III, and controls were discriminated, and sera were analyzed as indicated in Fig. 1 and Supplemental Fig. 1. a MMP-2 levels in the serum of individual patients according to achalasia type. b MMP-9 levels in analyzed sera. c Ratios of MMP-9 versus MMP-2 regarding to types of achalasia. d Ratios of activated versus proMMP-9 monomer forms in the defined types. For all types of achalasia the mean levels ±SD are provided, *p < 0.05; **p < 0.01; ***p < 0.001