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. 2018 Nov 7;12:396. doi: 10.3389/fncel.2018.00396

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Copyright © 2018 Chen, Wu, Gu, Zhou, Ye, Zhang, Pan, Wang, Wei, Zhu, Naman, Mak, Carlier, Cui and Han.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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A10E significantly prevented surgery-induced impairment of recognition in the early stage. The 1st NOR test was performed on days 5–7 post-operation. (A) In the training session of the 1st NOR test, treatments did not significantly change the recognition index in aged mice. (B) In the retention session of the 1st NOR test, A10E treatment at 0.4–0.6 mg/kg significantly prevented the reduction of recognition index in surgery-treated mice (n = 8). ^#p < 0.05 vs. the control group, **p < 0.01 vs. the surgery group (Dunn’s multiple comparison test).