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Kaplan-Meyer analysis of the event nephrotic syndrome during the disease course. The analysis shows that patients with properdin dependent C3 convertase stabilizing activity against spontaneous (A, Group −/+: sCSA−/sPCSA+) or FH-mediated (B, Group −/+: FH−CSA−/FH−PCSA+) decay, have higher risk to manifest nephrotic syndrome than patients with properdin-independent activities (A, Group +/+: sCSA+/sPCSA+; B, Group +/+: FH+CSA+/FH+PCSA+.
