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. 2018 Nov 21;9:4910. doi: 10.1038/s41467-018-07463-9

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Key signaling pathways involved in stomach development and gastric cancer. Statistical analyses of proteomic and transcriptomic data to identify differentially expressed genes (small lower squares on each gene symbol) or proteins (small upper squares on each gene symbol) in stomach development. Proteins differentially expressed and genes frequently mutated in DGC were compared to those in stomach development. Cross marks represent genes or proteins that are not detected in the study; solid lines represent cell membranes; dashed lines represent nuclear membranes; lines with solid arrows illustrate activation of downstream genes; lines with hammer-heads illustrate inhibition of downstream genes; gene symbols in cyan are upregulated in tumor (FDR < 0.05); italic gene symbols with underlines are top mutated genes in DGC