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. 2018 Nov 8;2018:5076271. doi: 10.1155/2018/5076271

(a).

Reference Radiation SAR Exposure time Model Temperature Effect
[157] RF 900 MHz 0.090 W/kg Long-term exposure Mice Mitochondrial DNA damage
[158] RF 900 MHz 2–5.7 W/kg Short-term exposure Human spermatozoa Constant No effect on mitochondria
[67] RF 850 MHz 1.46 W/kg Short-term exposure Human semen Room temperature Loss in sperm motility and viability
Increased ROS production
[133] RF 1.8 GHz 0.4–27.5 W/kg Short-term exposure Human spermatozoa Constant Mitochondrial ROS generation
Alterations of electron potential of mitochondrial membrane,
Oxidative DNA damage
[124] RF 900–1800 MHz Long-term exposure Rat Constant Lower sperm motility
Decreased GSH levels
Oxidative damage
[140] RF 900 MHz 0.9 W/kg Long-term exposure Rat Decreased antioxidant enzyme activity
Increased ROS production
Lipid peroxidation
Increased apoptosis
[144] MW 10 GHz 0.014 W/kg Long-term exposure Rat Spermatozoa cell cycle arrest
ROS production
[145] RF 900–1800–1900 MHz 0.9 W/kg Long-term exposure Rat Decreased antioxidant defences
Lipid peroxidation
[122] RF 900 MHz Long-term exposure Rat Decreased sperm count
Decreased antioxidant defences
Lipid peroxidation
[141] RF 900 MHz 0.66 W/kg Long-term exposure Rat Decreased sperm viability
Increased ROS production
Increased apoptosis
[156] RF 1800–1900 MHz Long-term exposure Human Mitochondrial damage
[154] RF 900 MHz pulsed 0.0516–0.0054 W/kg Long-term exposure Mice Alterations of electron potential of mitochondrial membrane
DNA damage
[146] RF 902.4 MHz 0.00516–0.0054 W/kg Long-term exposure Mice Decreased antioxidant defences
Lipid peroxidation
DNA damage
Decreased sperm count
[142] RF 900 MHz 0.0067 W/kg Long-term exposure Rat Constant Imbalance in total antioxidant capacity
Lipid peroxidation
[155] RF 1800 MHz 0.15 W/kg Short-term exposure Mouse germ cells Increased ROS production
DNA fragmentation
Oxidative DNA damage
[143] RF 1800 MHz 4 W/kg Short-term exposure Mouse germ cells Oxidative DNA damage
Increased autophagy