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. 2018 Nov 11;2018:7956503. doi: 10.1155/2018/7956503

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Copyright © 2018 Jingkun Lu et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

PMC Copyright notice

The FCN alters the expression of genes that are related to stress reaction, apoptosis and necrosis of cardiomyocytes, the inflammatory response. (a–f) mRNA levels of the proinflammatory cytokines TNF-α, IL-1β, stress cytokine p38, and the proapoptosis cytokines P53, Bax, and antiapoptosis cytokines Bcl-2, as well as Bcl-2/Bax ratio of the injured myocardium of each intervention group (n = 6 mice per group). Mean values ± SD. are represented. ^##P < 0.01 versus the control group, ^#P < 0.05 versus the control group, ∗∗P < 0.01 versus model, and ∗P < 0.05 versus model.