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. 2018 Nov 20;9:965. doi: 10.3389/fneur.2018.00965

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Copyright © 2018 Li, Li, Chopp, Venkat, Zacharek, Chen, Landschoot-Ward, Yan and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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ICH increases apoptosis, cardiac fibrosis and cardiomyocyte hypertrophy at 28 days after ICH; ICH does not affect DAP, MAP, and SAP compared to sham control mice. ICH in mice induces cardiac fibrosis and cardiomyocyte hypertrophy as well as apoptosis at 28 days after ICH (A) Terminal deoxynucleotidyl transferase (TUNEL) staining for cardiomyocyte apoptosis and quantitative data, scale bar, 20 μm; (B) Picro Sirius Red (PSR) staining and quantitative data for interstitial collagen fraction (ICF) and cardiomyocyte cross-sectional areas (MCSA) measurement at 28 days after ICH, scale bar, 20 μm. (C–E) (DAP), mean arterial pressure (MAP) and systolic arterial pressure (SAP) measurement at 1, 3, 7 days after ICH. Data are presented as mean ± SE, ^*p < 0.05 compared with sham control.