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. 2018 Nov 6;43(1):358–370. doi: 10.3892/ijmm.2018.3980

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Promotion of VEGF inhibited the effects of miR-138 upregulation on LDH activity of uterine endothelial cells in a co-culture with THP-1 cells. (A) Western blot analysis, and quantified levels of (B) Bax and (C) VEGF protein expression. (D) Cell growth, (E) LDH activity, (F) DAPI assay, (G) caspase-3 levels and (H) caspase-9 levels. ^##P<0.01 vs. control group; ^**P<0.01 vs. miR-138 group. miR, microRNA; Bax, B-cell lymphoma 2-associated X protein; VEGF, vascular endothelial growth factor; LDH, lactate dehydrogenase; miR-138, overexpression group; miR-138+VEGF, overexpression of miR-138 and VEGF.