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C8‐SA activates autophagy in wild‐type worms, but fails to increase autophagy in worms that are defective for the UPRmit (ubl‐5 mutants). Interestingly, the sole genetic ablation of ubl‐5 sufficed to induce autophagy monitored by increased LGG‐1::GFP levels. This reveals an unforeseen interaction between autophagy and the UPRmit
