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. 2018 Nov 28;24:61. doi: 10.1186/s10020-018-0059-9

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — miR-96 is predicted to regulate the MAPK signaling pathway via CACNG5. a prediction of target relationship between miR-96 and CACNG5 promoter region; b the effect of miR-96 on WT-CACNG5 and Mut-CACNG5 luciferase reporter plasmid; c the effect of CACNG5 on the expression of iNOS protein; d and e, effect of miR-96 on the expression of iNOS protein. ^*, P < 0.05 compared with the NC group. The test was repeated three times independently, and the non-paired t-test was used for comparison between the two groups; the one-way ANOVA analysis was used for comparison among multiple groups. miR-96, microRNA-96; CACNG5, calcium voltage-gated channel auxiliary subunit gamma 5; Wt, wild-type; Mut, mutant; NC, negative control