Skip to main content
. 2018 Nov 14;10(11):1757. doi: 10.3390/nu10111757

Figure 2.

Figure 2

Lipid profile. The rats were fed the STD or CAF diet for 9 weeks. Then, the rats in the STD and CAF groups were treated orally with RSV (50, 100 or 200 mg per kg of body weight) for 3 weeks. Total lipids in (A) eWAT, (B) liver and (C) calf skeletal muscle in mg for each g of tissue. (D) Expression in the liver of genes related to lipogenesis (Acc, Scd1 and Fas) and β-oxidation (Ppara and Cpt1b). (E) Expression in eWAT of genes related to lipogenesis (Acc, Scd1 and Fas) and β-oxidation (Ppara and Cpt1b). (F) Expression in calf skeletal muscle of genes related to β-oxidation (Cpt1b), mitochondrial biogenesis (Pgc1a) and thermogenesis (Ucp2 and Ucp3). Leptin gene expression in (G) eWAT and (H) rWAT. Data are expressed as the mean ± SEM, n = 6. * denotes p < 0.05, Student’s t-test comparing the CAF group to the STD group. # indicates p < 0.05 and ε p < 0.1, Student’s t-test comparing the RSV group to the CAF group. CAF: cafeteria diet; eWAT: epididymal white adipose tissue; RSV: resveratrol; rWAT: retroperitoneal white adipose tissue; STD: standard chow diet. Acc (acetyl-CoA carboxylase); Cpt1b (carnitine palmitoyltransferase 1b); Fas (fatty acid synthase); Pgc1a (peroxisome proliferator-activated receptor gamma coactivator 1-alpha), Ppara (peroxisome proliferator activated receptor alpha); Scd1 (stearoyl-CoA desaturase 1); Ucp2 (mitochondrial uncoupling protein 2); Ucp3 (mitochondrial uncoupling protein 3).