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. 2018 Aug 29;374(3):607–617. doi: 10.1007/s00441-018-2905-z

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© The Author(s) 2018

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 5 — S117A-FGF2 prevents Dox-induced ROS accumulation and oxidation of phosphatidylcholines. Neonatal rat cardiomyocytes were treated with Dox in the presence and absence of S117A-FGF-2 pre-treatment. a The effect of S117A-FGF2 on Dox-induced Dox upregulation as measured by the DCF-DA assay. b–d Levels of oxidized phosphatidylcholine species (HODA-PPC (PLPC); PAPC (–OOH); SLPC (–OH, –OOH)) measured by LC/MS/MS and normalized for protein concentration. The Y-axis in panels b–d shows the amount (ng) of OxPC per μg cardiomyocyte protein. In all panels, brackets denote groups significantly different from each other (n = 3 independent analyses)