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. 2014 Sep 2;19(9):13587–13602. doi: 10.3390/molecules190913587

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© 2014 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/3.0/).

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(A) Hydroxyl radical scavenging activity of VKP and VKCFR. (B) Protective effects of VKP and VKCFR pretreatment at different concentrations (1, 5, and 50 μM) on RCMEC exposed to 300 μM H₂O₂ in the MTT assay. (C) Effects of VKP and VKCFR treatment at different concentrations (1, 5, and 50 μM) on SOD enzymatic activity in RCMEC exposed to H₂O₂. (D) Effects of VKP and VKCFR treatment at different concentrations (1, 5 and 50 μM) with H₂O₂ exposure on RCMEC CAT enzymatic activity. (E) Effects of H₂O₂ exposure with VKP and VKCFR treatment at different concentrations (1, 5, and 50 μM) on RCMEC GSH-px enzymatic activity. Significant difference compared to the control at # p < 0.05, ## p < 0.01; significant differences compared to the negative control at * p < 0.05, ** p < 0.01, n = 6 in each group.