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. 2018 Nov 27;5:316. doi: 10.3389/fmed.2018.00316

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Copyright © 2018 Bennett, Reeves, Billman and Sturmberg.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The clinical changes and physiological mechanisms of allergic respiratory disease. Antigen-presenting cells (APCs) present allergen-derived peptides lodged within the MHC molecule to T cells within a T_H2 milieu, resulting in allergy mediated by eosinophils and IgE derived from plasma cells (transformed from activated B cells). PAMP, pathogen-associated molecular pattern; TLR, toll-like receptor; MHC, major histocompatibility complex; TCR, T cell receptor; IgE, immunoglobulin E; OX40L, CD252; B7, CD28; IL-4, interleukin-4; IL-5, interleukin-5; IL-13, interleukin-13.