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. 2018 Dec 4;9:335. doi: 10.1186/s13287-018-1083-y

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — Graphical abstract of our research. BMSCs could migrate to the injured lungs and inhibit silica-induced pulmonary fibrosis. The mechanism may be due to paracrine action rather than the potential of BMSCs to differentiate into alveolar epithelial cells