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. 2018 Nov 5;15(11):1399–1409. doi: 10.1080/15476286.2018.1536593

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© 2018 The Author(s). Published by Taylor & Francis.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited, and is not altered, transformed, or built upon in any way.

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Figure 2. — Identification of miRNAs binding to SSR tracts. (A) Number of miRNAs predicted to bind to disease-relevant SSRs showing 7-nt and 6-nt complementarity to SSRs within their seed regions. (B) Secondary structure representations of complexes between CUG repeats and miR-15a-5p and between CAG repeats and miR-370-3p. SSR tracts are highlighted in yellow; miRNA seed regions are highlighted in red. (C) Distribution of the number of predicted MREs for miRNAs interacting with SSRs in the 3ʹ UTRs of protein-coding transcripts and in ncRNAs (lncRNAs, pseudogenes and circRNAs). Each point represents a single RNA; only RNAs with a minimum of 10 MREs are shown. (D) The density of predicted MREs was calculated as the number of MREs per 1 kb of the analyzed RNAs with multiple (minimum of 10) MREs and is depicted as the mean value with the SEM (symbols with bars).