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. 2018 Dec 7;9:5235. doi: 10.1038/s41467-018-07552-9

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Clinical and molecular features of cyclin-activated HCC. a t-SNE plots depicting the classification of HCC from the LICA-FR and TCGA cohorts based on their transcriptional profiles. Tumors harboring CCNA2 or CCNE1 activating alterations are indicated with a color code. b Clinical characteristics, driver genes and deregulated pathways associated with CCN-HCC in the LICA-FR (top) and TCGA (bottom) cohorts. c Overall survival in CCN-HCC as compared with other HCC in the LICA-FR cohort. Only HCC with curative resection (R0) were included