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. 2018 Dec 3;12:865. doi: 10.3389/fnins.2018.00865

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Copyright © 2018 Li, Gao, Kang, Zhang, Zhao, Zhai, Huang, Yang, Sun and Wang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Rapamycin attenuated spinal cord ischemia-reperfusion induced apoptosis. (A). These cells were stained with TUNEL and DAPI. The merged picture shows colocolization of apoptotic and nuclear markers. Scare bar = 50 μm. (B).The bar graph shows rapamycin treatment rescued neurons from apoptosis at 24 h after SCIR. Data were shown as mean ± SD, n = 6. The numbers of apoptotic cells were counted in three sections of ischemic spinal cord. ^∗p < 0.05, ^∗∗p < 0.01 vs. Sham; ^#p < 0.05, ^##p < 0.01. (C) Bar graphs show the rapamycin-treated mice have significantly higher BMS scores than the vehicle-treated mice at 24 h after injury. Data were shown as mean ± SD, n = 16. ^∗p < 0.05, ^∗∗p < 0.01 vs. Sham; ^#p < 0.05, ^##p < 0.01.