Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2018 Sep 26;36(6):401–407. doi: 10.1136/acupmed-2016-011353

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2018. All rights reserved. No commercial use is permitted unless otherwise expressly granted.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/

PMC Copyright notice

Effect of EA on Glu content and expression of NMDAR2A and NMDAR2B protein. (A) Glu content in the CA1 area of the hippocampus in each group, measured by ELISA. (B) Protein expression of NMDAR2A and NMDAR2B in the CA1 area of the hippocampus in each group, examined by Western blotting. (C,D) Quantitative indices of protein expression of NR2A and NR2B, derived from figure 2B. #P<0.05 versus control group; *P<0.05 versus MCAO group. EA, electroacupuncture; Glu, glutamate; MCAO, middle cerebral artery occlusion; NMDAR2A, N-methyl-D-asparticacid receptors 2A; NMDAR2B, N-methyl-D-asparticacid receptors 2B.