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. 2018 Dec 4;9:1403. doi: 10.3389/fphar.2018.01403

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Copyright © 2018 Xu, Hua, Zhong, Li, Xu, Cai, Mao and Lu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Inhibition of oxidative stress and apoptosis by RES-HSA-NPs (RES-NPs) in rats following ischemia-reperfusion. (A) RES-NPs attenuated oxidative stress in brain after I/R. Quantified (n = 4) MDA levels of the cerebral cortex from I/R side after 24 h reperfusion. The RES-NPs (20 mg/kg) treatment had significantly decreased cortex MDA levels compared with the vechicle (^∗∗P < 0.01 vs. sham, ^##P < 0.01 vs. vehicle, ANOVA). (B) Representative photomicrographs showing yellow-brown TUNEL staining cells (pointed by white arrows) in penumbral region of brain sections from Sham, vehicle and RES-NPs treated rats after 72 h reperfusion (n = 4). Scale bars = 100 μm. (C) Quantification of the TUNEL-staining cells. ^∗P < 0.05 vs. sham, ^#P < 0.05 vs. vehicle.