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. 2018 Nov 5;73(1):102–111. doi: 10.1161/HYPERTENSIONAHA.118.12162

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© 2018 The Authors.

Hypertension is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited and is not used for commercial purposes.

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Figure 1. — Mice with myeloid cell–restricted MR (mineralocorticoid receptor) deficiency display improved cardiac function and remodeling after myocardial infarction (MI). A, Representative sections from MR^flox and MR^LysMCre infarcted hearts and infarct size. B, Representative left ventricular (LV) pressure-volume loops measured in vivo with conductance catheter in sham-operated MR^flox (gray) and MR^LysMCre (black) mice and in MR^flox (orange) and MR^LysMCre (blue) mice with MI. C, LV systolic pressure (LVSP), LV end-diastolic pressure (LVEDP), LV end-systolic and end-diastolic volumes; LV ejection fraction, LV maximal rate of pressure rise (LV dP/dt_max), maximal rate of pressure decline (LV dP/dt_min), and LV dP/dt_max divided by instantaneous pressure (IP) and the time constant of LV pressure isovolumic decay (Tau). Mean±SEM (n=14–16). *P<0.01 vs MR^flox.