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. 2018 Dec 6;9:1395. doi: 10.3389/fphar.2018.01395

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Copyright © 2018 Alencar, Pimentel-Coelho, Montes, da Silva, Mendes, Montagnoli, Silva, Vasques, Rosado-de-Castro, Gutfilen, Cunha, Fraga, Silva, Martins, Ferreira, Mendes-Otero, Trachez, Sudo and Zapata-Sudo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effects of the hMSC therapy in the pulmonary vascular extracellular matrix deposition. (A) Picrosirius red staining. (B) Shows the Western blot analyses of RAGE in lungs from all animal groups. GAPDH was used for normalization. (C) Perivascular collagen area, and (D) quantification of RAGE expression. Each column and bar represent the mean ± SEM (n = 5–7 mice per group). ^∗P < 0.05 compared with normoxia group; ^†P < 0.05 compared with SuH group treated with vehicle. Ordinary one-way ANOVA with multiple comparisons. hMSC, human mesenchymal stem-cell; SuH, SU5416/hypoxia; RAGE, receptor for advanced glycation end products; CSA, cross section area.