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. 2018 Oct 1;132(24):e35–e46. doi: 10.1182/blood-2018-02-831982

Table 1.

Correlations with scores per study in class I (in vivo thrombus formation) or class III (in vitro thrombus formation) for different vascular beds and different triggers

Groups N 3-point P 5-point P
Kendall τ Kendall τ
Compared with carotis (1-4)
 Mesentery 6-8 53 0.80 <.001 0.53 <.001
 Cremaster 9-11 27 0.89 <.001 0.66 <.001
Compared with FeCl3 (1, 6, 9)
 Laser 8, 11 38 0.78 <.001 0.50 .001
 Photochemical 2, 7, 10 27 0.67 <.001 0.55 .001
 Ligation and compression 3, 5 23 0.59 .003 0.52 .005
Compared with class III (30)
 Carotis 1-4 77 0.57 <.001 0.49 <.001
 Mesentery 6-8 65 0.77 <.001 0.65 <.001
 Cremaster 9-11 35 0.80 <.001 0.64 <.001
 FeCl3 1, 6, 9 99 0.67 <.001 0.62 <.001
 Laser 8, 11 36 0.86 <.001 0.56 .001
 Photochemical 2, 7, 10 15 0.26 .325 0.46 .056
 Ligation and compression 3, 5 24 0.87 <.001 0.70 <.001
 Collagen/epinephrine 20 14 0.71 .006 0.25 .286

Indicated are correlations across genes for similar genetically modified mice, used for studies in multiple classes, when scored on 3-point or 5-point scales. N refers to number of comparisons (genes) made. Comparison for indicated groups (injury modes, in italic) are indicated relative to carotid models (groups 1-4), FeCl3 injury models (groups 1, 6, 9), or in vitro studies (group 30).