Table 4:

Odds ratios (OR) and 95% confidence intervals (CI) for esophageal cancer associated with alcohol type, expressed as the origin of ethanol origi risk in Kenya: effects of drinking intensity, duration and quitting, stratified by ever-tobacco use Associations by alcohol type, in ever-alcohol drinkers

Categorical		Numbers of cases / controls	Median ethanol/day Cases, controls	OR (95% CI), Model 2a + adjusted for grams of ethanol categories
% ethanol (g) from busaa	0-<10%	44/56	43, 19	1
	10-<50%	128/58	71, 94	3.0 (1.6, 5.5)
	≥50%	136/68	53, 36	2.4 (1.3, 4.4)
% ethanol (g) from chang’aa	0-<5%	48/69	30, 25	1
	5–24%	84/56	111, 98	1.8 (1.0, 3.5)
	≥ 25%	178/57	51, 32	2.9 (1.7, 5.1)
% ethanol (g) from commercial beer	0	197/81	41, 28	1
	>0-<50	81/52	103, 89	0.6 (0.3, 1.0)
	≥50%	32/49	107, 35	0.3 (0.2, 0.6)
% ethanol (g) from spirits	0	255/125	52, 32	1
	>0-<10	34/16	125,127	1.4 (0.6, 3.0)
	≥10%	21/41	103, 55	0.3 (0.2, 0.6)
Continuous (not adjusted for each other):		Median % in cases, controls	Median % in cases, controls	Per 10% increase in ethanol from each type of drink**
		Men	Women
% ethanol from busaa		44, 32	50, 65	1.05 (0.98, 1.13)
% ethanol from chang’aa		28, 14	35, 16	1.16 (1.07, 1.27)b
% ethanol from commercial beer		32, 0	0, 0	0.85 (0.78, 0.93)
% ethanol from spirits		0, 0	0, 0	0.75 (0.60, 0.92)

^aAs specified in Table 3

^bPersists when ethanol g/day is adjusted as linear effect