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. 2018 Dec 1;32(23-24):1537–1549. doi: 10.1101/gad.318436.118

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© 2018 Yu et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 2. — TR4 and NCoR1 significantly overlap in genome-wide binding site distribution. (A) Venn diagram of TR4, TR2, and NCoR1 ChIP-seq peaks in human erythroleukemia K562 cells. (B) Representative NCoR1, TR2, and TR4 overlapping peaks in the β-globin locus. (C) The top enriched gene ontology terms extracted from the TR4, NCoR1-1, and NCoR1-2 overlapping peak-associated genes.