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. 2018 Dec 18;15:345. doi: 10.1186/s12974-018-1384-1

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — TBI acute neuroinflammation network schematic. Model variables include resting microglia (mr), M1-like microglia (M1), M2-like microglia (M2), interleukin(IL)-1 (IL1), IL-12 (IL12), IL-4 (IL4), IL-10 (IL10), tissue damage (D), and type 2 T-helper cells (Th2). Model components appearing next to pathways stimulate (+) or inhibit (−) the correspoding reaction