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. 2018 Dec 13;9:1059. doi: 10.3389/fneur.2018.01059

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Copyright © 2018 Zeng, Wang, Guo, Zheng, Wang, Wu, Li, Wu, Chen, Xu, Wang and Lin.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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SNX27 haploinsufficiency prevents neuronal death and caspase-3 activation after SCI. (A) Representative images of the lesion center in the spinal cords from Snx27^+/+ mice and Snx27^+/− mice 7 days post injury, Scale bar = 200 μm. (B) NeuN⁺ cell quantification of the lesion center in the spinal cords from Snx27^+/+ mice and Snx27^+/− mice 7 days post injury, n = 4. (C) Western blot analysis of cleaved caspase-3 levels in the spinal cord of Snx27^+/+ mice and Snx27^+/− mice 3 days post injury. (D) Quantitative analysis of cleaved caspase-3 expression in the spinal cord of Snx27^+/+ mice and Snx27^+/− mice 3 days post injury, n = 4. Values are expressed as mean ± SEM and were evaluated by Student's independent sample t-test. *p < 0.05, **p < 0.01.