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Possible molecular mechanisms for tenascin-C (TNC) to induce pathological conditions underlying delayed cerebral ischemia after subarachnoid hemorrhage (SAH).
BBB: Blood-brain barrier; IL: interleukin; MAPK: mitogen-activated protein kinase; NF-κB: nuclear factor-kappa B; PDGF: platelet-derived growth factor; PDGFR: PDGF receptor; TLR4: Toll-like receptor 4.
