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. 2018 Dec 21;12:147–156. doi: 10.2147/OTT.S182537

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© 2019 Hu et al. This work is published and licensed by Dove Medical Press Limited

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A p38 agonist ameliorated the inhibitory effect of PTENP1 on glioma cell proliferation.

Notes: (A) The proliferative abilities of PTENP1-transfected SHG44 and U251 cells after P79350 treatment were examined with the EdU incorporation assay. (B and C) The CCK-8 assay was used to examine the proliferative abilities of PTENP1-transfected SHG44 and U251 cells after P79350 treatment. (D–F) Flow cytometry was used to analyze the cell cycle distribution of glioma cells after different treatments. ^**P<0.01 vs control or NC group, ^*P<0.05 vs control or NC group, and ^#P<0.05 vs PTENP1 group.

Abbreviations: CCK-8, Cell Counting Kit-8; NC, negative control.

A p38 agonist ameliorated the inhibitory effect of PTENP1 on glioma cell proliferation.

Notes: (A) The proliferative abilities of PTENP1-transfected SHG44 and U251 cells after P79350 treatment were examined with the EdU incorporation assay. (B and C) The CCK-8 assay was used to examine the proliferative abilities of PTENP1-transfected SHG44 and U251 cells after P79350 treatment. (D–F) Flow cytometry was used to analyze the cell cycle distribution of glioma cells after different treatments. ^**P<0.01 vs control or NC group, ^*P<0.05 vs control or NC group, and ^#P<0.05 vs PTENP1 group.

Abbreviations: CCK-8, Cell Counting Kit-8; NC, negative control.