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. 2018 Nov 23;17(1):613–620. doi: 10.3892/etm.2018.7007

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Copyright: © Ju et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — ACKR4 regulates CCL21 level in NPC tumor tissue. The intratumoral levels of (A) CCL21, (B) CCL19, (C) CCL25 and (D) CXCL13 were measured in tumors with different levels of ACKR4 expression. The concentration of CCL21 was evidently reduced by ACKR4 overexpression in NPC tumor tissue. ***P<0.001 and ****P<0.0001. NS, no significant difference. ACKR4, atypical chemokine receptor 4; NPC, nasopharyngeal carcinoma; WT, wild-type; EV, empty vector; KD, knockdown; OE, overexpression; CCL, C-C motif chemokine ligand; CXCL, C-X-C motif chemokine ligand.