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. 2018 Nov 22;23(1):328–339. doi: 10.1111/jcmm.13934

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© 2018 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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TES inhibited the calcineurin‐NFAT axis in murine hypertrophic hearts. A, Western blot of the total and phosphorylated protein concentrations of NFATc4, NFATc2, and NFATc1 in murine hearts in the indicated groups. B, Western blot of the total and phosphorylated protein concentration of MEK1/2, AKT, ERK1/2, P38, and JNK1/2 in murine hearts in the indicated groups. C and D, Representative Western blot and quantification of NFATc3 and MCIP1.4 protein levels in murine cytoplasm and nucleus in the indicated groups. (n = 4, *P < 0.05 vs GFP/sham; ^# P < 0.05 vs GFP/AB)